cytochrome cells
英
美
网络 胞色细胞
双语例句
- Cloning and Protein Release of Cytochrome C in the Apoptosis Cells of Silkworm, Bombyx mori
家蚕细胞色素C基因的克隆及其蛋白在家蚕凋亡细胞中的释放 - Influence of Cytochrome C on Apoptosis of Spodoptera Litura Cells in Cell-free System
细胞色素C在无细胞体系中对斜纹夜蛾细胞凋亡的影响 - Objective To investigate the effects of hemorrhagic shock on the structure and function of mitochondrial DNA ( mtDNA) encoding Cytochrome Oxidase genes in intestinal epithelial cells.
目的探讨失血性休克对大鼠肠上皮细胞线粒体DNA编码细胞色素氧化酶基因的损伤作用。 - Role of cytochrome C in ATP depletion-induced apoptosis in renal tubular epithelial cells
细胞色素C在ATP耗竭诱导的肾小管上皮细胞凋亡中的作用 - Objective: To investigate the effects of experimental varicocele on mitochondria calcium and cytochrome C of the epididymal cells in adolescent rats.
目的：探讨外科所致的精索静脉曲张对青春期大鼠附睾细胞线粒体Ca2+及细胞色素C的影响。 - Cytochrome oxidase reaction area of capillary mitochondria ( Ae) was smaller, whereas volume density of enzyme reaction ( Vve) was bigger than those of type ⅱ alveolar cells.
毛细血管线粒体细胞色素氧化酶阳性反应面积比Ⅱ型肺泡细胞小，但酶反应体密度大。 - The Changes of Cytochrome C Oxidase Gene ⅱ and Mitochondrial Membrane Potential in Smooth Muscle Cells of Antrum in Liver Cirrhosis Rats
肝硬化大鼠胃窦平滑肌细胞色素氧化酶Ⅱ基因及线粒体膜电位的变化 - Conclusion Cytochrome C release from mitochondria in ATP depleted renal tubular epithelial cells plays an essential role in initiating Caspase-dependent apoptosis.
结论肾小管上皮细胞ATP耗竭时，线粒体细胞色素C的释放在介导Caspase依赖的细胞凋亡通路中发挥重要作用。 - And cytochrome c was released from the mitochondria to the cytosol. CONCLUSION: VES could induce apoptosis of human gastric carcinoma SGC-7901 cells through mitochondrial pathway by Bax.
结论：VES可能通过Bax蛋白来启动线粒体凋亡途径，诱导SGC-7901细胞发生凋亡。 - Effect of opening of mitochondrial ATP-sensitive K~+ channel on the distribution of cytochrome C and on proliferation of human pulmonary arterial smooth muscle cells in hypoxia
缺氧肺动脉平滑肌细胞中线粒体ATP敏感钾通道开放对细胞色素C的分布及细胞增殖的作用 - Study on damage of genes of mitochondrial DNA encoding cytochrome oxidase of intestinal epithelial cells induced by hemorrhagic shock in rats
失血性休克大鼠小肠上皮细胞线粒体DNA、COX基因损伤的研究 - Conclusion Bcl-2 gene transfer was able to decrease the expression of cytochrome C, caspase-8 and caspase-3 and to protect myocardial cells against apoptosis in mice heart transplantation rejection.
结论心脏移植排斥反应中移植心肌细胞色素C、caspase-8和caspase-3表达均明显上升，Bcl-2基因转染抗心脏移植排斥反应中心肌细胞凋亡作用可能与其下调细胞色素C、caspase-8和caspase-3的表达有关。 - Results Inhibition of survivin by mutant survivin cDNA could induce apoptosis, increase caspase-3 activity, cleave PARP and promote cytochrome C release in gastric cancer cells. Inhibition of survivin also caused mitotic catastrophe in gastric cancer cells.
结果Cys84Ala可通过抑制野生型生存素基因的功能诱导胃癌细胞凋亡，增加caspase3活性，促进PARP裂解和细胞色素C的释放，亦可导致胃癌细胞发生有丝分裂障碍而死亡。 - Effect of hyperbaric oxygenation therapy on the expression of Bcl-2, Bax and cytochrome C in nerve cells of neonatal rats with hypoxic-ischemic brain damage
高压氧治疗对新生大鼠缺氧缺血性脑损伤模型神经细胞胞浆Bcl-2Bax和细胞色素C表达的影响 - Objective To study the changes of cytochrome C oxidase gene ⅱ mRNA ( CO ⅱ mRNA) and mitochondrial membrane potential ( MMP) in smooth muscle cells of antrum in liver cirrhosis rats.
目的研究肝硬化大鼠胃窦平滑肌细胞色素氧化酶Ⅱ基因（COⅡgene）及线粒体膜电位的变化，探讨肝硬化胃动力障碍机制。 - The level of ATP, cytochrome C, and energy charge in hepatic cells were applied to evaluate the function of mitochondria.
分离肝细胞线粒体，监测肝细胞线粒体内ATP水平、细胞色素C含量和能荷。 - Cytochrome c Can Induce Programmed Cell Death in Plant Cells
细胞色素c能诱导植物细胞编程性死亡 - Immunocytochemical method analysis showed that the protein expression of cytochrome C and caspase-9 were up-regulated in HeLa cells treated with curcumin.
免疫细胞化学分析发现HeLa细胞中CytochromeC、caspase-9的表达随姜黄素浓度的升高而增强。 - Furthermore, the release of cytochrome c from mitochondria was detected using confocal microscopy in HeLa cells treated with B1.
同时B1作用HeLa细胞后，通过共聚焦显微镜可观察到细胞色素C从线粒体释放。 - Moreover, cytochrome c oxidase activity rapidly decreased in fiber cells after rotenone treatment and polyphenoloxidase activity reduced by more than 80% in green cotton fiber after thiourea treatment. 4.
在鱼藤酮处理下，纤维细胞中的细胞色素c氧化酶的活性迅速降低，而在硫脲各浓度处理下，绿色棉纤维中的多酚氧化酶活性均降低了80%以上。 - The increase of protein level of cytochrome c in the cytosol and the caspase-3 activation of FL cells after TBT exposure demonstrated the important roles that both cytochrome c and caspase-3 played in TBT-induced apoptosis.
TBT暴露可导致FL细胞胞液中细胞色素c蛋白水平的升高和caspase-3活化，表明细胞色素c和caspase-3在TBT诱导的FL细胞凋亡中起到了重要作用。